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(9)   

• Imaging the effects of methylphenidate on brain dopamine: new model on its therapeutic actions for attention-deficit/hyperactivity disorder.
  Nora D Volkow and Gene-Jack Wang and Joanna S Fowler and Yu-Shin Ding Biol Psychiatry57(11):1410--1415Jun2005. ritalin increases DA to salient stims (likely phasic) .
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• D1 and D2 dopamine-receptor modulation of striatal glutamatergic signaling in striatal medium spiny neurons.
  D. James Surmeier and Jun Ding and Michelle Day and Zhongfeng Wang and Weixing Shen Trends Neurosci30(5):228--235May2007.
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• L-dopa impairs learning, but spares generalization, in Parkinson's disease.
  Daphna Shohamy and Catherine E Myers and Kindiya D Geghman and Jacob Sage and Mark A Gluck Neuropsychologia44(5):774--784(2006)
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• Therapeutic doses of amphetamine or methylphenidate differentially increase synaptic and extracellular dopamine.
  W. K. Schiffer and N. D. Volkow and J. S. Fowler and D. L. Alexoff and J. Logan and S. L. Dewey Synapse59(4):243--251Mar2006. ritalin and amphetamine both increase synaptic (phasic) DA to same degree (both amp increases extracellular DA much more) .
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• Dopaminergic activities in the human striatum: rostrocaudal gradients of uptake sites and of D1 and D2 but not of D3 receptor binding or dopamine.
  M. A. Piggott and E. F. Marshall and N. Thomas and S. Lloyd and J. A. Court and E. Jaros and D. Costa and R. H. Perry and E. K. Perry Neuroscience90(2):433--445May1999.
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• Effects of catecholamine uptake blockers in the caudate-putamen and subregions of the medial prefrontal cortex of the rat.
  M. S. Mazei and C. P. Pluto and B. Kirkbride and E. A. Pehek Brain Res936(1-2):58--67May2002. DAT blockade preferentially increases BG rel to PFC DA. .
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• Endocannabinoid-mediated rescue of striatal LTD and motor deficits in Parkinson's disease models.
  Anatol C Kreitzer and Robert C Malenka Nature445(7128):643--647Feb2007.
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• Selective elimination of glutamatergic synapses on striatopallidal neurons in Parkinson disease models.
  Michelle Day and Zhongfeng Wang and Jun Ding and Xinhai An and Cali A Ingham and Andrew F Shering and David Wokosin and Ema Ilijic and Zhuoxin Sun and Allan R Sampson and Enrico Mugnaini and Ariel Y Deutch and Susan R Sesack and Gordon W Arbuthnott and D. James Surmeier Nat Neurosci9(2):251--259Feb2006.
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• Inhibition of Dopamine Release Via Presynaptic D2 Receptors: Time Course and Functional Characteristics In Vivo
  M. Benoit-Marand and E. Borrelli and F. Gonon Journal of Neuroscience(2001) D2 presynaptic autoinhibiton. stimulated MFB, evoked DA relase. 100 Hz stim linearly inhibited further release. Stim mimicking burst (4 pulses @ 15 Hz) enh...D2 presynaptic autoinhibiton. stimulated MFB, evoked DA relase. 100 Hz stim linearly inhibited further release. Stim mimicking burst (4 pulses @ 15 Hz) enhanced extracellular DA and inhibited further release, this autoinhibition was maximal between 150-300 ms and disappeared within 600 ms. -->autoinhibition elicited by one burst transiently attenuates further dopamine release elicited by tonic activity.. haldol should block this effect! .
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