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Calmodulin kinase signaling in heart: an intriguing candidate target for therapy of myocardial dysfunction and arrhythmias.

. Pharmacol. Ther. 106 (1): 39--55 (April 2005)

Abstract

The multifunctional Ca$^2+$/calmodulin (CaM)-dependent protein kinase II (CaMKII) has emerged as a proarrhythmic and procardiomyopathic signal in a wide range of structural heart diseases. This review discusses CaMKII structure and function and recent evidence implicating CaMKII inhibition as a potential strategy for treating myocardial dysfunction and arrhythmias in the setting of structural heart disease.

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DOI:
10.1016/j.pharmthera.2004.11.002
URL:
BibTeX key:
Ande_2005_39
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