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Calsequestrin determines the functional size and stability of cardiac intracellular calcium stores: Mechanism for hereditary arrhythmia.

, , , , , and . Proc. Natl. Acad. Sci. U. S. A., 100 (20): 11759--11764 (September 2003)
DOI: 10.1073/pnas.1932318100

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Repolarization abnormalities and afterdepolarizations in a canine model of sudden cardiac death., , , , , , , , , and 1 other author(s). Am J Physiol Regul Integr Comp Physiol, 295 (5): R1463--R1472 (November 2008)Calsequestrin determines the functional size and stability of cardiac intracellular calcium stores: Mechanism for hereditary arrhythmia., , , , , and . Proc. Natl. Acad. Sci. U. S. A., 100 (20): 11759--11764 (September 2003)Mechanisms of impaired calcium handling underlying subclinical diastolic dysfunction in diabetes., , , , , , , , and . Am J Physiol Regul Integr Comp Physiol, 293 (5): R1787--R1797 (November 2007)Modulation of SR Ca release by luminal Ca and calsequestrin in cardiac myocytes: effects of CASQ2 mutations linked to sudden cardiac death., , , , , , , , , and 1 other author(s). Biophys J, 95 (4): 2037--2048 (August 2008)Abnormal calcium signaling and sudden cardiac death associated with mutation of calsequestrin., , , , , , , , , and . Circ. Res., 94 (4): 471--477 (March 2004)Enhanced ryanodine receptor-mediated calcium leak determines reduced sarcoplasmic reticulum calcium content in chronic canine heart failure., , , , , and . Biophys J, 93 (11): 4083--4092 (December 2007)Modulation of cytosolic and intra-sarcoplasmic reticulum calcium waves by calsequestrin in rat cardiac myocytes., , , , , , and . J. Physiol., 561 (Pt 2): 515--524 (December 2004)Abnormal intrastore calcium signaling in chronic heart failure., , , , , , , , , and 2 other author(s). Proc. Natl. Acad. Sci. U. S. A., 102 (39): 14104--14109 (September 2005)Triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytes., , , , , , , and . Circ. Res., 96 (6): 651--658 (April 2005)Synergistic interactions between Ca$^2+$ entries through L-type Ca$^2+$ channels and Na$^+$-Ca$^2+$ exchanger in normal and failing rat heart., , , , and . J. Physiol., 567 (Pt 2): 493--504 (September 2005)