Abstract
Perinatal hypoxic-ischemic cerebral injury, secondary to interruption of placental blood flow that results in cerebral palsy (CP), is a rare event. The ability to link an intrapartum event to subsequent CP should include a history of a sentinel event during labor, followed by the delivery of a depressed acidemic infant, and the subsequent evolution of neonatal encephalopathy, systemic organ injury, and acute neuroimaging abnormalities.
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