Congenital bilateral perisylvian syndrome was diagnosed in a two-year- old boy with signs of pseudobulbar and diplegic cerebral palsy presenting on MRI a polymicrogyric appearance of the perisylvian regions. He was born from a monochorionic bi-amniotic twin pregnancy complicated by twin-twin transfusion syndrome and death of the co-twin between the 16th and 18th weeks of gestation. Ventricular enlargement and hepatic hyperechogenic lesions were seen during his sonographic intra-uterine follow-up. The authors suggest that ischaemic injury occurred in this patient as a consequence of acute haemodynamic changes induced by the death of his co-twin.
%0 Journal Article
%1 Bogaert1996
%A Bogaert, P. Van
%A Donner, C.
%A David, P.
%A Rodesch, F.
%A Avni, E. F.
%A Szliwowski, H. B.
%D 1996
%J Dev Med Child Neurol
%K Cerebral Aqueduct; Palsy; Child, Preschool; Female; Fetal Death; Fetofetal Transfusion; Humans; Male; Paralysis; Pregnancy; Syndrome; Twins, Monozygotic
%N 2
%P 166--170
%T Congenital bilateral perisylvian syndrome in a monozygotic twin with intra-uterine death of the co-twin.
%V 38
%X Congenital bilateral perisylvian syndrome was diagnosed in a two-year- old boy with signs of pseudobulbar and diplegic cerebral palsy presenting on MRI a polymicrogyric appearance of the perisylvian regions. He was born from a monochorionic bi-amniotic twin pregnancy complicated by twin-twin transfusion syndrome and death of the co-twin between the 16th and 18th weeks of gestation. Ventricular enlargement and hepatic hyperechogenic lesions were seen during his sonographic intra-uterine follow-up. The authors suggest that ischaemic injury occurred in this patient as a consequence of acute haemodynamic changes induced by the death of his co-twin.
@article{Bogaert1996,
abstract = {Congenital bilateral perisylvian syndrome was diagnosed in a two-year- old boy with signs of pseudobulbar and diplegic cerebral palsy presenting on MRI a polymicrogyric appearance of the perisylvian regions. He was born from a monochorionic bi-amniotic twin pregnancy complicated by twin-twin transfusion syndrome and death of the co-twin between the 16th and 18th weeks of gestation. Ventricular enlargement and hepatic hyperechogenic lesions were seen during his sonographic intra-uterine follow-up. The authors suggest that ischaemic injury occurred in this patient as a consequence of acute haemodynamic changes induced by the death of his co-twin.},
added-at = {2014-07-19T19:06:34.000+0200},
author = {Bogaert, P. Van and Donner, C. and David, P. and Rodesch, F. and Avni, E. F. and Szliwowski, H. B.},
biburl = {https://www.bibsonomy.org/bibtex/299b05f9ff369422c0ad04e872fb8207b/ar0berts},
groups = {public},
interhash = {4319c0722ceec1ad0b76726f0e08e18b},
intrahash = {99b05f9ff369422c0ad04e872fb8207b},
journal = {Dev Med Child Neurol},
keywords = {Cerebral Aqueduct; Palsy; Child, Preschool; Female; Fetal Death; Fetofetal Transfusion; Humans; Male; Paralysis; Pregnancy; Syndrome; Twins, Monozygotic},
month = Feb,
number = 2,
pages = {166--170},
pmid = {8603784},
timestamp = {2014-07-19T19:06:34.000+0200},
title = {Congenital bilateral perisylvian syndrome in a monozygotic twin with intra-uterine death of the co-twin.},
username = {ar0berts},
volume = 38,
year = 1996
}