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Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1

R. Verhaak, K. Hoadley, E. Purdom, V. Wang, Y. Qi, M. Wilkerson, C. Miller, L. Ding, T. Golub, J. Mesirov, G. Alexe, M. Lawrence, M. O'Kelly, P. Tamayo, B. Weir, S. Gabriel, W. Winckler, S. Gupta, L. Jakkula, H. Feiler, J. Hodgson, C. James, J. Sarkaria, C. Brennan, A. Kahn, P. Spellman, R. Wilson, T. Speed, J. Gray, M. Meyerson, G. Getz, C. Perou, and D. Hayes. Cancer Cell, 17 (1): 98 - 110 (2010)
DOI: https://doi.org/10.1016/j.ccr.2009.12.020

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Todd Templeton

Koraljka Golub

Catarina Alice Todd

Yulia Golub

Todd Sepke

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COT drives resistance to RAF inhibition through MAP kinase pathway reactivationC. Johannessen, J. Boehm, S. Kim, S. Thomas, L. Wardwell, L. Johnson, C. Emery, N. Stransky, A. Cogdill, J. Barretina and 25 other author(s). Nature, 468 (7326): 968--972 (Dec 16, 2010)Comprehensive genomic characterization defines human glioblastoma genes and core pathwaysT. Network, R. McLendon, A. Friedman, D. Bigner, E. Van Meir, D. Brat, G. M. Mastrogianakis, J. Olson, T. Mikkelsen, N. Lehman and 222 other author(s). Nature, (September 2008)Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1R. Verhaak, K. Hoadley, E. Purdom, V. Wang, Y. Qi, M. Wilkerson, C. Miller, L. Ding, T. Golub, J. Mesirov and 23 other author(s). Cancer Cell, 17 (1): 98--110 (2010)Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1R. Verhaak, K. Hoadley, E. Purdom, V. Wang, Y. Qi, M. Wilkerson, C. Miller, L. Ding, T. Golub, J. Mesirov and 23 other author(s). Cancer Cell, 17 (1): 98 - 110 (2010)Metagenes and molecular pattern discovery using matrix factorizationJ. Brunet, P. Tamayo, R. Golub, and P. Mesirov. Proc. Natl. Acad. Sci. U. S. A., (2004)Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profilesA. Subramanian, P. Tamayo, K. Mootha, S. Mukherjee, L. Ebert, a. Gillette, A. Paulovich, L. Pomeroy, R. Golub, S. Lander and 1 other author(s). Proc. Natl. Acad. Sci. U. S. A., (2005)Class prediction and discovery using gene expression data.D. Slonim, P. Tamayo, J. Mesirov, T. Golub, and E. Lander. RECOMB, page 263-272. ACM, (2000)Metagenes and molecular pattern discovery using matrix factorizationJ. Brunet, P. Tamayo, T. Golub, and J. Mesirov. PNAS, 101 (12): 4164-4169 (2004)Initial genome sequencing and analysis of multiple myelomaM. Chapman, M. Lawrence, J. Keats, K. Cibulskis, C. Sougnez, A. Schinzel, C. Harview, J. Brunet, G. Ahmann, M. Adli and 40 other author(s). Nature, 471 (7339): 467--472 (April 2012)Interpreting Patterns of Gene Expression with Self-Organizing Maps: Methods and Application to Hematopoietic DifferentiationP. Tamayo, D. Slonim, J. Mesirov, Q. Zhu, S. Kitareewan, E. Dmitrovsky, E. Lander, and T. Golub. Proceedings of the National Academy of Sciences, USA, (1999)

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Disambiguation of "Golub, Todd"

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Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1

Please choose a person to relate this publication to

Todd Templeton

Koraljka Golub

Catarina Alice Todd

Yulia Golub

Todd Sepke

Other publications of authors with the same name

Disambiguation

BibSonomy

Disambiguation of "Golub, Todd"

copydeleteadd this publication to your clipboardcommunity posthistory of this postURLDOIBibTeXEndNoteAPAChicagoDIN 1505HarvardMSOffice XML Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1

Please choose a person to relate this publication to

Todd Templeton

Koraljka Golub

Catarina Alice Todd

Yulia Golub

Todd Sepke

Other publications of authors with the same name

Disambiguation

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URL
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Integrated Genomic Analysis Identifies Clinically Relevant Subtypes of Glioblastoma Characterized by Abnormalities in PDGFRA, IDH1, EGFR, and NF1