Prader–Willi Syndrome (PWS) is a complex neurogenetic disorder caused by loss of the paternal 15q11.2–q13 locus, due to deletion (DEL), maternal uniparental disomy (mUPD), or imprinting center defects. Individuals with mUPD have up to 60% risk of developing psychosis in early adulthood. Given the increasing evidence for white matter abnormalities in psychotic disorders, we investigated white matter microstructure in children and adolescents with PWS, with a particular emphasis on the DEL and mUPD subtypes. Please contact the library to request a copy of this article - http://bit.ly/1Xyazai