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Upon intranasal VSV infection astrocytes in the olfactory bulb are important IFN-beta producers that protect from lethal encephalitis

, , , , , , , , , , and . J Virol, 89 (5): 2731-2738 (2015)

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Type I Interferon Receptor Signaling of Neurons and Astrocytes Regulates Microglia Activation during Viral Encephalitis, , , , , , , , , and 7 other author(s). Cell Rep, 25 (1): 118-129.e4 (October 2018)IFN-beta deficiency results in fatal or demyelinating disease in C57BL/6 mice infected with Theiler's murine encephalomyelitis viruses, , , , , , , , , and 2 other author(s). Front Immunol, (February 2022)Host strategies against virus entry via the olfactory system., , and . Virulence, 2 (4): 367-370 (2011)Interferon-beta expression and type I interferon receptor signaling of hepatocytes prevent hepatic necrosis and virus dissemination in Coxsackievirus B3-infected mice, , , , , , , , , and 12 other author(s). PLOS Pathog, 14 (8): 1-23 (August 2018)The type I interferon response bridles rabies virus infection and reduces pathogenicity., , , , and . J Neurovirol, 17 (4): 353-367 (November 2011)Concomitant TLR/RLH Signaling of Radioresistant and Radiosensitive Cells Is Essential for Protection against Vesicular Stomatitis Virus Infection, , , , , , , , , and 2 other author(s). J Immunol, 193 (6): 3045-3054 (2014)Upon intranasal VSV infection astrocytes in the olfactory bulb are important IFN-beta producers that protect from lethal encephalitis, , , , , , , , , and 1 other author(s). J Virol, 89 (5): 2731-2738 (2015)Interferon-beta expression and type I interferon receptor signaling of hepatocytes prevent hepatic necrosis and virus dissemination in Coxsackievirus B3-infected mice, , , , , , , , , and 32 other author(s). PLOS Pathog, 14 (8): 1-23 (August 2018)Matrix protein mediated shutdown of host cell metabolism limits vesicular stomatitis virus-induced interferon-alpha responses to plasmacytoid dendritic cells, , , and . Immunobiology, 212 (9-10): 887-894 (2007)Hepatitis C Virus Stimulates Murine CD8alpha-Like Dendritic Cells to Produce Type I Interferon in a TRIF-Dependent Manner, , , , , , , and . PLoS Pathog, 12 (7): e1005736 (2016)